nhaliday + gwern   123

Complexity no Bar to AI - Gwern.net
Critics of AI risk suggest diminishing returns to computing (formalized asymptotically) means AI will be weak; this argument relies on a large number of questionable premises and ignoring additional resources, constant factors, and nonlinear returns to small intelligence advantages, and is highly unlikely. (computer science, transhumanism, AI, R)
created: 1 June 2014; modified: 01 Feb 2018; status: finished; confidence: likely; importance: 10
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april 2018 by nhaliday
Are Sunk Costs Fallacies? - Gwern.net
But to what extent is the sunk cost fallacy a real fallacy?
Below, I argue the following:
1. sunk costs are probably issues in big organizations
- but maybe not ones that can be helped
2. sunk costs are not issues in animals
3. sunk costs appear to exist in children & adults
- but many apparent instances of the fallacy are better explained as part of a learning strategy
- and there’s little evidence sunk cost-like behavior leads to actual problems in individuals
4. much of what we call sunk cost looks like simple carelessness & thoughtlessness
ratty  gwern  analysis  meta-analysis  faq  biases  rationality  decision-making  decision-theory  economics  behavioral-econ  realness  cost-benefit  learning  wire-guided  marginal  age-generation  aging  industrial-org  organizing  coordination  nature  retention  knowledge  iq  education  tainter  management  government  competition  equilibrium  models  roots  chart 
december 2017 by nhaliday
Fish on Friday | West Hunter
There are parts of Europe, Switzerland and Bavaria for example, that are seriously iodine deficient. This used to be a problem. I wonder if fish on Friday ameliorated it: A three-ounce serving size of cod provides your body with 99 micrograms of iodine, or 66% of the recommended amount per day.

Thinking further, it wasn’t just Fridays: there were ~130 days a years when the Catholic Church banned flesh.

Gwern on modern iodine-deficiency: https://westhunt.wordpress.com/2017/10/28/fish-on-friday/#comment-97137
population surveys indicate lots of people are iodine-insufficient even in the US or UK where the problem should’ve been permanently solved a century ago
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october 2017 by nhaliday
Of Mice and Men | West Hunter
It’s not always easy figuring out how a pathogen causes disease. There is an example in mice for which the solution was very difficult, so difficult that we would probably have failed to discover the cause of a similarly obscure infectious disease in humans.

Mycoplasma pulmonis causes a chronic obstructive lung disease in mice, but it wasn’t easy to show this. The disease was first described in 1915, and by 1940, people began to suspect Mycoplasma pulmonis might be the cause. But then again, maybe not. It was often found in mice that seemed healthy. Pure cultures of this organism did not consistently produce lung disease – which means that it didn’t satisfy Koch’s postulates, in particular postulate 1 (The microorganism must be found in abundance in all organisms suffering from the disease, but should not be found in healthy organisms.) and postulate 3 (The cultured microorganism should cause disease when introduced into a healthy organism.).

Well, those postulates are not logic itself, but rather a useful heuristic. Koch knew that, even if lots of other people don’t.

This respiratory disease of mice is long-lasting, but slow to begin. It can take half a lifetime – a mouse lifetime, that is – and that made finding the cause harder. It required patience, which means I certainly couldn’t have done it.

Here’s how they solved it. You can raise germ-free mice. In the early 1970s, researchers injected various candidate pathogens into different groups of germ-free mice and waited to see which, if any, developed this chronic lung disease. It was Mycoplasma pulmonis , all right, but it had taken 60 years to find out.

It turned out that susceptibility differed between different mouse strains – genetic susceptibility was important. Co-infection with other pathogens affected the course of the disease. Microenvironmental details mattered – mainly ammonia in cages where the bedding wasn’t changed often enough. But it didn’t happen without that mycoplasma, which was a key causal link, something every engineer understands but many MDs don’t.

If there was a similarly obscure infectious disease of humans, say one that involved a fairly common bug found in both the just and the unjust, one that took decades for symptoms to manifest – would we have solved it? Probably not.

Cooties are everywhere.

gay germ search: https://westhunt.wordpress.com/2013/07/21/of-mice-and-men/#comment-15905
It’s hard to say, depends on how complicated the path of causation is. Assuming that I’m even right, of course. Some good autopsy studies might be fruitful – you’d look for microanatomical brain differences, as with nartcolepsy. Differences in gene expression, maybe. You could look for a pathogen – using the digital version of RDA (representational difference analysis), say on discordant twins. Do some old-fashioned epidemiology. Look for marker antibodies, signs of some sort of immunological event.

Do all of the above on gay rams – lots easier to get started, much less whining from those being vivisected.

Patrick Moore found the virus causing Kaposi’s sarcoma without any funding at all. I’m sure Peter Thiel could afford a serious try.
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september 2017 by nhaliday
trees are harlequins, words are harlequins — bayes: a kinda-sorta masterpost
lol, gwern: https://www.reddit.com/r/slatestarcodex/comments/6ghsxf/biweekly_rational_feed/diqr0rq/
> What sort of person thinks “oh yeah, my beliefs about these coefficients correspond to a Gaussian with variance 2.5″? And what if I do cross-validation, like I always do, and find that variance 200 works better for the problem? Was the other person wrong? But how could they have known?
> ...Even ignoring the mode vs. mean issue, I have never met anyone who could tell whether their beliefs were normally distributed vs. Laplace distributed. Have you?
I must have spent too much time in Bayesland because both those strike me as very easy and I often think them! My beliefs usually are Laplace distributed when it comes to things like genetics (it makes me very sad to see GWASes with flat priors), and my Gaussian coefficients are actually a variance of 0.70 (assuming standardized variables w.l.o.g.) as is consistent with field-wide meta-analyses indicating that d>1 is pretty rare.
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august 2017 by nhaliday
A combined analysis of genetically correlated traits identifies 107 loci associated with intelligence | bioRxiv
We apply MTAG to three large GWAS: Sniekers et al (2017) on intelligence, Okbay et al. (2016) on Educational attainment, and Hill et al. (2016) on household income. By combining these three samples our functional sample size increased from 78 308 participants to 147 194. We found 107 independent loci associated with intelligence, implicating 233 genes, using both SNP-based and gene-based GWAS. We find evidence that neurogenesis may explain some of the biological differences in intelligence as well as genes expressed in the synapse and those involved in the regulation of the nervous system.


Finally, using an independent sample of 6 844 individuals we were able to predict 7% of intelligence using SNP data alone.
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july 2017 by nhaliday
Polygenic transmission disequilibrium confirms that common and rare variation act additively to create risk for autism spectrum disorders : Nature Genetics : Nature Research
Autism spectrum disorder (ASD) risk is influenced by common polygenic and de novo variation. We aimed to clarify the influence of polygenic risk for ASD and to identify subgroups of ASD cases, including those with strongly acting de novo variants, in which polygenic risk is relevant. Using a novel approach called the polygenic transmission disequilibrium test and data from 6,454 families with a child with ASD, we show that polygenic risk for ASD, schizophrenia, and greater educational attainment is over-transmitted to children with ASD. These findings hold independent of proband IQ. We find that polygenic variation contributes additively to risk in ASD cases who carry a strongly acting de novo variant. Lastly, we show that elements of polygenic risk are independent and differ in their relationship with phenotype. These results confirm that the genetic influences on ASD are additive and suggest that they create risk through at least partially distinct etiologic pathways.

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july 2017 by nhaliday
The Genomic Health Of Ancient Hominins | bioRxiv
On a broad scale, hereditary disease risks are similar for ancient hominins and modern-day humans, and the GRS percentiles of ancient individuals span the full range of what is observed in present day individuals. In addition, there is evidence that ancient pastoralists may have had healthier genomes than hunter-gatherers and agriculturalists. We also observed a temporal trend whereby genomes from the recent past are more likely to be healthier than genomes from the deep past.

Gwern has interesting take (abstract is misleading): https://twitter.com/gwern/status/871061144152178688

here it is in conclusion (and cf Figure 3A):
The genomic health of ancient individuals appears to have improved over time (Figure 3B). This calls into question the idea that genetic load has been increasing in human populations (Lynch 2016). However, there exists a perplexing pattern: ancient individuals who lived within the last few thousand years have healthier genomes, on average, than present day humans.

After controlling for age, BMI, and other variables, knee OA prevalence was 2.1-fold higher (95% confidence interval, 1.5–3.1) in the postindustrial sample than in the early industrial sample. Our results indicate that increases in longevity and BMI are insufficient to explain the approximate doubling of knee OA prevalence that has occurred in the United States since the mid-20th century. Knee OA is thus more preventable than is commonly assumed, but prevention will require research on additional independent risk factors that either arose or have become amplified in the postindustrial era.
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june 2017 by nhaliday
Genomic analysis of family data reveals additional genetic effects on intelligence and personality | bioRxiv
Using Extended Genealogy to Estimate Components of Heritability for 23 Quantitative and Dichotomous Traits: http://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1003520
Pedigree- and SNP-Associated Genetics and Recent Environment are the Major Contributors to Anthropometric and Cardiometabolic Trait Variation: http://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1005804

Missing Heritability – found?: https://westhunt.wordpress.com/2017/02/09/missing-heritability-found/
There is an interesting new paper out on genetics and IQ. The claim is that they have found the missing heritability – in rare variants, generally different in each family.

Some of the variants, the ones we find with GWAS, are fairly common and fitness-neutral: the variant that slightly increases IQ confers the same fitness (or very close to the same) as the one that slightly decreases IQ – presumably because of other effects it has. If this weren’t the case, it would be impossible for both of the variants to remain common.

The rare variants that affect IQ will generally decrease IQ – and since pleiotropy is the norm, usually they’ll be deleterious in other ways as well. Genetic load.

Happy families are all alike; every unhappy family is unhappy in its own way.: https://westhunt.wordpress.com/2017/06/06/happy-families-are-all-alike-every-unhappy-family-is-unhappy-in-its-own-way/
It now looks as if the majority of the genetic variance in IQ is the product of mutational load, and the same may be true for many psychological traits. To the extent this is the case, a lot of human psychological variation must be non-adaptive. Maybe some personality variation fulfills an evolutionary function, but a lot does not. Being a dumb asshole may be a bug, rather than a feature. More generally, this kind of analysis could show us whether particular low-fitness syndromes, like autism, were ever strategies – I suspect not.

It’s bad new news for medicine and psychiatry, though. It would suggest that what we call a given type of mental illness, like schizophrenia, is really a grab-bag of many different syndromes. The ultimate causes are extremely varied: at best, there may be shared intermediate causal factors. Not good news for drug development: individualized medicine is a threat, not a promise.

see also comment at: https://pinboard.in/u:nhaliday/b:a6ab4034b0d0

So the big implication here is that it's better than I had dared hope - like Yang/Visscher/Hsu have argued, the old GCTA estimate of ~0.3 is indeed a rather loose lower bound on additive genetic variants, and the rest of the missing heritability is just the relatively uncommon additive variants (ie <1% frequency), and so, like Yang demonstrated with height, using much more comprehensive imputation of SNP scores or using whole-genomes will be able to explain almost all of the genetic contribution. In other words, with better imputation panels, we can go back and squeeze out better polygenic scores from old GWASes, new GWASes will be able to reach and break the 0.3 upper bound, and eventually we can feasibly predict 0.5-0.8. Between the expanding sample sizes from biobanks, the still-falling price of whole genomes, the gradual development of better regression methods (informative priors, biological annotation information, networks, genetic correlations), and better imputation, the future of GWAS polygenic scores is bright. Which obviously will be extremely helpful for embryo selection/genome synthesis.

The argument that this supports mutation-selection balance is weaker but plausible. I hope that it's true, because if that's why there is so much genetic variation in intelligence, then that strongly encourages genetic engineering - there is no good reason or Chesterton fence for intelligence variants being non-fixed, it's just that evolution is too slow to purge the constantly-accumulating bad variants. And we can do better.

The surprising implications of familial association in disease risk: https://arxiv.org/abs/1707.00014
As Greg Cochran has pointed out, this probably isn’t going to work. There are a few genes like BRCA1 (which makes you more likely to get breast and ovarian cancer) that we can detect and might affect treatment, but an awful lot of disease turns out to be just the result of random chance and deleterious mutation. This means that you can’t easily tailor disease treatment to people’s genes, because everybody is fucked up in their own special way. If Johnny is schizophrenic because of 100 random errors in the genes that code for his neurons, and Jack is schizophrenic because of 100 other random errors, there’s very little way to test a drug to work for either of them- they’re the only one in the world, most likely, with that specific pattern of errors. This is, presumably why the incidence of schizophrenia and autism rises in populations when dads get older- more random errors in sperm formation mean more random errors in the baby’s genes, and more things that go wrong down the line.

The looming crisis in human genetics: http://www.economist.com/node/14742737
Some awkward news ahead
- Geoffrey Miller

Human geneticists have reached a private crisis of conscience, and it will become public knowledge in 2010. The crisis has depressing health implications and alarming political ones. In a nutshell: the new genetics will reveal much less than hoped about how to cure disease, and much more than feared about human evolution and inequality, including genetic differences between classes, ethnicities and races.

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june 2017 by nhaliday
Pearson correlation coefficient - Wikipedia
what does this mean?: https://twitter.com/GarettJones/status/863546692724858880
deleted but it was about the Pearson correlation distance: 1-r
I guess it's a metric


A less misleading way to think about the correlation R is as follows: given X,Y from a standardized bivariate distribution with correlation R, an increase in X leads to an expected increase in Y: dY = R dX. In other words, students with +1 SD SAT score have, on average, roughly +0.4 SD college GPAs. Similarly, students with +1 SD college GPAs have on average +0.4 SAT.

this reminds me of the breeder's equation (but it uses r instead of h^2, so it can't actually be the same)

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may 2017 by nhaliday
Reversal of Fortune | West Hunter
“particularly in the fetus”. You’d think so, but people have looked at Dutch draftees who were in the womb during the famine of 1944. They found no effects of famine exposure on Ravens scores at age 19. Schizophrenia doubled, though. Schiz also doubled in the Chinese cohort exposed to the Great Leap Forward famine.

Cohort Profile: The Dutch Hunger Winter Families Study: https://academic.oup.com/ije/article/36/6/1196/814573
Nutrition and Mental Performance: https://sci-hub.bz/10.1126/science.178.4062.708
Schizophrenia after prenatal exposure to the Dutch Hunger Winter of 1944-1945: https://sci-hub.bz/10.1001/archpsyc.1992.01820120071010
Prenatal famine exposure and cognition at age 59 years: https://sci-hub.bz/10.1093/ije/dyq261

You might be right. There is reason to suspect that prenatal exposure to alcohol is far riskier in some populations than others – in particular populations that have limited historical exposure to alcohol. Fetal Alcohol Syndrome (FAS) is very rare in France, for example – yet they drink, I’m told.

The kind of conservatism that shows up politically doesn’t have any predictive value. In other words, liars and morons. They’re why God made baseball bats. Once upon a time, I said this: “The American right doesn’t have room for anyone who knows jack shit about anything, or whose predictions have ever come true.” I’ll stick with that.

full quote here: http://www.rpgcodex.net/forums/index.php?threads/planescape-torment-problems.9208/
The American right doesn't have room for anyone who knows jack shit about anything, or whose predictions have ever come true. Of course they're all liars. In the words of one of their semi-prominent members, himself plenty despicable: "Science, logic, rational inquiry, thoughtful reflection, mean nothing to them. It's all posturing and moral status games and sucking up to halfwits like GWB and clinging to crackpot religion, and of course amoral careerism. " I think my correspondent forgot to mention their propensity for eating shit and rolling around in their own vomit, but nobody's perfect.

I’ve mused that it’s generally believed that iodine benefits females more than males, and the timing of iodization in the US matches up reasonably well with the rise of feminism…
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may 2017 by nhaliday
Syphilis in Renaissance Europe: rapid evolution of an introduced sexually transmitted disease?
When syphilis first appeared in Europe in 1495, it was an acute and extremely unpleasant disease. After only a few years it was less severe than it once was, and it changed over the next 50 years into a milder, chronic disease. The severe early symptoms may have been the result of the disease being introduced into a new host population without any resistance mechanisms, but the change in virulence is most likely to have happened because of selection favouring milder strains of the pathogen.
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may 2017 by nhaliday
Intersection of diverse neuronal genomes and neuropsychiatric disease: The Brain Somatic Mosaicism Network
Towards explaining non-shared-environment effects on intelligence, psychiatric disorders, and other cognitive traits - developmental noise such as post-conception mutations in individual cells or groups of cells
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may 2017 by nhaliday
Soft sweeps are the dominant mode of adaptation in the human genome | bioRxiv
Detection of 2000 instances of recent human selection, half of which are specific to individual human populations, and many of which affect the central nervous system. Note this doesn’t cover polygenic selection, so it’s a very loose lower bound on how much recent human evolution there has been.

How Sweeps Get Soft: https://westhunt.wordpress.com/2011/09/29/how-sweeps-get-soft/
A puzzling finding from the search for selection in humans is the large number of apparently selected variants versus the apparent absence of high frequency selected regions and regions that have become fixed in our species.

Cochran, John Hawks, and I were involved in some of this work, and we all thought immediately without any discussion that the intermediate frequency sweeps must reflect heterozygote advantage.
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may 2017 by nhaliday
The Roman State and Genetic Pacification - Peter Frost, 2010
- Table 1 is a good summary, but various interesting tidbits throughout
main points:
- latrones reminds me of bandit-states, Big Men in anthropology, and Rome's Indo-European past
- started having trouble recruiting soldiers, population less martial
- Church opposition to State violence, preferred to 'convert enemies by prayer'
- a Christian could use violence 'only to defend others and not for self-defense'
- Altar of Victory was more metaphorical than idolatrous, makes its removal even more egregious


should read:
BANDITS IN THE ROMAN EMPIRE: http://sci-hub.tw/http://academic.oup.com/past/article-abstract/105/1/3/1442375/BANDITS-IN-THE-ROMAN-EMPIRE
Bandits in the Roman Empire: Myth and reality: https://historicalunderbelly.files.wordpress.com/2012/12/thoma-grunewald-bandits-in-the-roman-empire-myth-and-reality-2004.pdf

What Difference Did Christianity Make?: http://sci-hub.tw/https://www.jstor.org/stable/4435970
Author(s): Ramsay Mac Mullen

The extent of this impact I test in five areas. The first two have to do with domestic relations: sexual norms and slavery. The latter three have to do with matters in which public authorities were more involved: gladiatorial shows, judicial penalties, and corruption.

Clark/Frost Domestication: https://westhunt.wordpress.com/2013/05/14/clarkfrost-domestication/
Thinking about the response of the pacified and submission Roman population to barbarian invaders immediately brings to mind the response of contemporary North Americans and Atlantic Europeans to barbarian invaders. It reads just the same: “welcome new neighbor!”

What about the Eastern empire? They kept the barbarians out for a few centuries longer in the European half, but accounts of the loss of the Asian provinces show the Clark/Frost pattern, a pacified submissive population hardly contesting the invasion of Islam (Jenkins 2008, 2010). The new neighbors simply walked in and took over. The downfall of the Western Roman empire reads much like the downfall of the Asian and North African parts of the empire. It is certainly no accident that the Asian provinces were the heartland of Christianity.

This all brings up an interesting question: what happened in East Asia over the same period? No one to my knowledge has traced parallels with the European and Roman experience in Japan or China. Is the different East Asian trajectory related to the East Asian reluctance to roll over, wag their tails, and welcome new barbarian neighbors?

gwern in da comments
“empires domesticate their people”
Greg said in our book something like “for the same reason that farmers castrate their bulls”
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may 2017 by nhaliday
Educational Romanticism & Economic Development | pseudoerasmus


Did Nations that Boosted Education Grow Faster?: http://econlog.econlib.org/archives/2012/10/did_nations_tha.html
On average, no relationship. The trendline points down slightly, but for the time being let's just call it a draw. It's a well-known fact that countries that started the 1960's with high education levels grew faster (example), but this graph is about something different. This graph shows that countries that increased their education levels did not grow faster.

Where has all the education gone?: http://citeseerx.ist.psu.edu/viewdoc/download?doi=




The Case Against Education: What's Taking So Long, Bryan Caplan: http://econlog.econlib.org/archives/2015/03/the_case_agains_9.html

The World Might Be Better Off Without College for Everyone: https://www.theatlantic.com/magazine/archive/2018/01/whats-college-good-for/546590/
Students don't seem to be getting much out of higher education.
- Bryan Caplan

College: Capital or Signal?: http://www.economicmanblog.com/2017/02/25/college-capital-or-signal/
After his review of the literature, Caplan concludes that roughly 80% of the earnings effect from college comes from signalling, with only 20% the result of skill building. Put this together with his earlier observations about the private returns to college education, along with its exploding cost, and Caplan thinks that the social returns are negative. The policy implications of this will come as very bitter medicine for friends of Bernie Sanders.

Doubting the Null Hypothesis: http://www.arnoldkling.com/blog/doubting-the-null-hypothesis/

Is higher education/college in the US more about skill-building or about signaling?: https://www.quora.com/Is-higher-education-college-in-the-US-more-about-skill-building-or-about-signaling
ballpark: 50% signaling, 30% selection, 20% addition to human capital
more signaling in art history, more human capital in engineering, more selection in philosophy

Econ Duel! Is Education Signaling or Skill Building?: http://marginalrevolution.com/marginalrevolution/2016/03/econ-duel-is-education-signaling-or-skill-building.html
Marginal Revolution University has a brand new feature, Econ Duel! Our first Econ Duel features Tyler and me debating the question, Is education more about signaling or skill building?

Against Tulip Subsidies: https://slatestarcodex.com/2015/06/06/against-tulip-subsidies/




Most American public school kids are low-income; about half are non-white; most are fairly low skilled academically. For most American kids, the majority of the waking hours they spend not engaged with electronic media are at school; the majority of their in-person relationships are at school; the most important relationships they have with an adult who is not their parent is with their teacher. For their parents, the most important in-person source of community is also their kids’ school. Young people need adult mirrors, models, mentors, and in an earlier era these might have been provided by extended families, but in our own era this all falls upon schools.

Caplan gestures towards work and earlier labor force participation as alternatives to school for many if not all kids. And I empathize: the years that I would point to as making me who I am were ones where I was working, not studying. But they were years spent working in schools, as a teacher or assistant. If schools did not exist, is there an alternative that we genuinely believe would arise to draw young people into the life of their community?


It is not an accident that the state that spends the least on education is Utah, where the LDS church can take up some of the slack for schools, while next door Wyoming spends almost the most of any state at $16,000 per student. Education is now the one surviving binding principle of the society as a whole, the one black box everyone will agree to, and so while you can press for less subsidization of education by government, and for privatization of costs, as Caplan does, there’s really nothing people can substitute for it. This is partially about signaling, sure, but it’s also because outside of schools and a few religious enclaves our society is but a darkling plain beset by winds.

This doesn’t mean that we should leave Caplan’s critique on the shelf. Much of education is focused on an insane, zero-sum race for finite rewards. Much of schooling does push kids, parents, schools, and school systems towards a solution ad absurdum, where anything less than 100 percent of kids headed to a doctorate and the big coding job in the sky is a sign of failure of everyone concerned.

But let’s approach this with an eye towards the limits of the possible and the reality of diminishing returns.

The real reason the left would support Moander: the usual reason. because he’s an enemy.

I have a problem in thinking about education, since my preferences and personal educational experience are atypical, so I can’t just gut it out. On the other hand, knowing that puts me ahead of a lot of people that seem convinced that all real people, including all Arab cabdrivers, think and feel just as they do.

One important fact, relevant to this review. I don’t like Caplan. I think he doesn’t understand – can’t understand – human nature, and although that sometimes confers a different and interesting perspective, it’s not a royal road to truth. Nor would I want to share a foxhole with him: I don’t trust him. So if I say that I agree with some parts of this book, you should believe me.


Caplan doesn’t talk about possible ways of improving knowledge acquisition and retention. Maybe he thinks that’s impossible, and he may be right, at least within a conventional universe of possibilities. That’s a bit outside of his thesis, anyhow. Me it interests.

He dismisses objections from educational psychologists who claim that studying a subject improves you in subtle ways even after you forget all of it. I too find that hard to believe. On the other hand, it looks to me as if poorly-digested fragments of information picked up in college have some effect on public policy later in life: it is no coincidence that most prominent people in public life (at a given moment) share a lot of the same ideas. People are vaguely remembering the same crap from the same sources, or related sources. It’s correlated crap, which has a much stronger effect than random crap.

These widespread new ideas are usually wrong. They come from somewhere – in part, from higher education. Along this line, Caplan thinks that college has only a weak ideological effect on students. I don’t believe he is correct. In part, this is because most people use a shifting standard: what’s liberal or conservative gets redefined over time. At any given time a population is roughly half left and half right – but the content of those labels changes a lot. There’s a shift.

I put it this way, a while ago: “When you think about it, falsehoods, stupid crap, make the best group identifiers, because anyone might agree with you when you’re obviously right. Signing up to clear nonsense is a better test of group loyalty. A true friend is with you when you’re wrong. Ideally, not just wrong, but barking mad, rolling around in your own vomit wrong.”
You just explained the Credo quia absurdum doctrine. I always wondered if it was nonsense. It is not.
Someone on twitter caught it first – got all the way to “sliding down the razor blade of life”. Which I explained is now called “transitioning”

What Catholics believe: https://theweek.com/articles/781925/what-catholics-believe
We believe all of these things, fantastical as they may sound, and we believe them for what we consider good reasons, well attested by history, consistent with the most exacting standards of logic. We will profess them in this place of wrath and tears until the extraordinary event referenced above, for which men and women have hoped and prayed for nearly 2,000 years, comes to pass.

According to Caplan, employers are looking for conformity, conscientiousness, and intelligence. They use completion of high school, or completion of college as a sign of conformity and conscientiousness. College certainly looks as if it’s mostly signaling, and it’s hugely expensive signaling, in terms of college costs and foregone earnings.

But inserting conformity into the merit function is tricky: things become important signals… because they’re important signals. Otherwise useful actions are contraindicated because they’re “not done”. For example, test scores convey useful information. They could help show that an applicant is smart even though he attended a mediocre school – the same role they play in college admissions. But employers seldom request test scores, and although applicants may provide them, few do. Caplan says ” The word on the street… [more]
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april 2017 by nhaliday
PsycARTICLES - Is education associated with improvements in general cognitive ability, or in specific skills?
Results indicated that the association of education with improved cognitive test scores is not mediated by g, but consists of direct effects on specific cognitive skills. These results suggest a decoupling of educational gains from increases in general intellectual capacity.

look at Model C for the coefficients

How much does education improve intelligence? A meta-analysis: https://psyarxiv.com/kymhp
Intelligence test scores and educational duration are positively correlated. This correlation can be interpreted in two ways: students with greater propensity for intelligence go on to complete more education, or a longer education increases intelligence. We meta-analysed three categories of quasi-experimental studies of educational effects on intelligence: those estimating education-intelligence associations after controlling for earlier intelligence, those using compulsory schooling policy changes as instrumental variables, and those using regression-discontinuity designs on school-entry age cutoffs. Across 142 effect sizes from 42 datasets involving over 600,000 participants, we found consistent evidence for beneficial effects of education on cognitive abilities, of approximately 1 to 5 IQ points for an additional year of education. Moderator analyses indicated that the effects persisted across the lifespan, and were present on all broad categories of cognitive ability studied. Education appears to be the most consistent, robust, and durable method yet to be identified for raising intelligence.

three study designs: control for prior IQ, exogenous policy change, and school age cutoff regression discontinuity

It’s surprising that there isn’t much of a fadeout (p11) – half of the effect size is still there by age 70 (?!). That wasn’t what I expected. Maybe they’re being pulled upwards by smaller outlier studies – most of the bigger ones tend towards the lower end.

These gains are hollow, as they acknowledge in the discussion. Examples:
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march 2017 by nhaliday
Cat Ladies | West Hunter
If toxo naturally can make people like cat piss, it’s already preadapted to become (with suitable genetic engineering) the model system for many kinds of infectious behavior modifiers.
west-hunter  sapiens  parasites-microbiome  disease  toxo-gondii  neuro  psychiatry  nature  discussion  commentary  gwern  medicine  🌞  embodied  scitariat  hmm  biotech  biohacking  retrofit  public-health  model-organism  pro-rata  epidemiology  drugs  pharma 
march 2017 by nhaliday
Epidemiology, epigenetics and the ‘Gloomy Prospect’: embracing randomness in population health research and practice | International Journal of Epidemiology | Oxford Academic
Despite successes in identifying causes, it is often claimed that there are missing additional causes for even reasonably well-understood conditions such as lung cancer and coronary heart disease. Several lines of evidence suggest that largely chance events, from the biographical down to the sub-cellular, contribute an important stochastic element to disease risk that is not epidemiologically tractable at the individual level. Epigenetic influences provide a fashionable contemporary explanation for such seemingly random processes. Chance events—such as a particular lifelong smoker living unharmed to 100 years—are averaged out at the group level. As a consequence population-level differences (for example, secular trends or differences between administrative areas) can be entirely explicable by causal factors that appear to account for only a small proportion of individual-level risk. In public health terms, a modifiable cause of the large majority of cases of a disease may have been identified, with a wild goose chase continuing in an attempt to discipline the random nature of the world with respect to which particular individuals will succumb.

choice quote:
"With the perception (in my view exaggerated) that genome-wide association studies (GWASs) have failed to deliver on initial expectations,5 the next phase of enhanced risk prediction will certainly shift to ‘epigenetics’6,7—the currently fashionable response to any question to which you do not know the answer."
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march 2017 by nhaliday
The Morality of Sperm Donation - Gwern.net
There is a third benefit. Surprisingly, sperm donor-assisted pregnancies result in 1/5th the number of birth defects as pregnancies in general. (The CDC tells me that the defect rate is 1 in 33 or ~3%, and that birth defects in 2006 directly killed 5,819 infants.) Much of this large benefit stems from the paternal age effect - older fathers’ sperm result in more birth defects, lowered IQ, linked to autism, etc. To the extent that a sperm donor donating displaces, at the margin, the conception of future offspring at an elder age, donation directly reduces birth defects and the other mentioned effects.

Indeed, given the mixed data on whether sperm count rates are falling with time and the more reliable data on the striking increases with paternal age of negative effects like birth defects or autism - perhaps due to increased mutations4 - it may be worthwhile even for non-donators to bank their sperm for later use. A quick perusal of one sperm bank’s prices suggests that a 20-year old could store a large sample of his sperm until he is 40 for ~$5000 with the ultimate artificial insemination adding <$4000 to the final cost; if this resulted in cutting the risk of his children being autistic by 30%, would it be worthwhile? Perhaps. It is worth considering.
ratty  gwern  hmm  sex  genetics  genetic-load  paternal-age  parenting  developmental  disease  data  effective-altruism  arbitrage  cost-benefit  analysis  faq  planning 
february 2017 by nhaliday
Holocene selection for variants associated with cognitive ability: Comparing ancient and modern genomes. | bioRxiv
- Michael Woodley


Human populations living in Eurasia during the Holocene experienced significant evolutionary change. It has been predicted that the transition of Holocene populations into agrarianism and urbanization brought about culture-gene co-evolution that favoured via directional selection genetic variants associated with higher general cognitive ability (GCA).
These observations are consistent with the expectation that GCA rose during the Holocene.
study  preprint  bio  sapiens  genetics  genomics  GWAS  antiquity  trends  iq  dysgenics  recent-selection  aDNA  multi  hn  commentary  gwern  enhancement  evolution  blowhards  behavioral-gen 
february 2017 by nhaliday
Embryo editing for intelligence - Gwern.net
My hunch is CRISPR/Cas9 will not play a big role in intelligence enhancement. You'd have to edit so many loci b/c of small effect sizes, increasing errors. Embryo selection is much more promising. Peoples with high avg genetic values, of course, have an in-built advantage there.
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february 2017 by nhaliday
Confounder Of The Day: How Sexy Your Parents Were | Slate Star Codex
- "paternal age effect" just a selection effect (men w/ issues end up having kids later due to difficulty finding a mate)
- one other suggested inconsistent explanation: spermatogenic selfish-gene effect
- interesting discussion of sperm freezing
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february 2017 by nhaliday
Funding the Reproducibility Crises as effective giving - Less Wrong Discussion
I had definitely noticed all the different nutrition, psychology, and biological initiatives like OSF or the Reproducibility Project, and how expensive they all are, but I didn't realize that they all owed their funding to a single source. I'm very glad Arnold is doing this, but I now feel more pessimistic about academia than when I assumed that the funding for all this was coming from a broad coalition of universities and nonprofits etc....
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january 2017 by nhaliday
Selection against variants in the genome associated with educational attainment
first direct, genotypic, longitudinal evidence I think?
fulltext: https://www.dropbox.com/s/9vq5t6urtu930xe/2017-kong.pdf

Epidemiological and genetic association studies show that genetics play an important role in the attainment of education. Here, we investigate the effect of this genetic component on the reproductive history of 109,120 Icelanders and the consequent impact on the gene pool over time. We show that an educational attainment polygenic score, POLY_EDU, constructed from results of a recent study is associated with delayed reproduction (P < 10^−100) and fewer children overall. _The effect is stronger for women and remains highly significant after adjusting for educational attainment._ Based on 129,808 Icelanders born between 1910 and 1990, we find that the average POLY_EDU has been declining at a rate of ∼0.010 standard units per decade, which is substantial on an evolutionary timescale. Most importantly, because POLY_EDU only captures a fraction of the overall underlying genetic component the latter could be declining at a rate that is two to three times faster.

- POLY_EDU has negative effect on RS for men, while EDU itself (or just controlling for POLY_EDU?) has positive effect
- also has some trends for height (0) and schizophrenia (-)

Natural selection making 'education genes' rarer, says Icelandic study: https://www.reddit.com/r/slatestarcodex/comments/5opugw/natural_selection_making_education_genes_rarer/
Gwern pretty pessimistic


The Marching Morons: https://westhunt.wordpress.com/2017/01/22/the-marching-morons/
There’s a new paper out on how the frequency of variants that affect educational achievement (which also affect IQ) have been changing over time in Iceland. Naturally, things are getting worse.

We don’t have all those variants identified yet, but from the fraction we do know and the rate of change, they estimate that genetic potential for IQ is dropping about 0.30 point per decade – 3 points per century, about a point a generation. In Iceland.

Sounds reasonable, in the same ballpark as demography-based estimates.

It would be interesting to look at moderately recent aDNA and see when this trend started – I doubt if has been going on very long. [ed.: I would guess since the demographic transition/industrial revolution, though, right?]

This is the most dangerous threat the human race faces.

Paper Review: Icelandic Dysgenics: http://www.unz.com/akarlin/paper-review-icelandic-dysgenics/
The main mechanism was greater age at first child, not total number of children (i.e. the clever are breeding more slowly).
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january 2017 by nhaliday
The High-Cost, High-Risk World of Modern Pet Care : slatestarcodex
What's particularly interesting about pet healthcare is, besides the usual insanity of many pet owners, is what it says about human healthcare. Pet healthcare is a libertarian ideal: vets require next to zero malpractice insurance, the FDA regulates all animal drugs and procedures far less than humans, pets live short lives compared to humans so any benefits are easy to observe and research (and the research itself is inherently far easier), insurance and HMOs and government programs are barely involved as most buyers pay cash, veterinarians themselves are generally in oversupply, etc. It's as close to an ideal perfectly competitive free market of healthcare as we'll ever see. And what's the result in America?
> The cost of veterinary care has risen even faster than the cost of human health care, more than doubling since 2000, according to the U.S. Bureau of Labor Statistics.
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january 2017 by nhaliday
Dehaene describes some fascinating and convincing evidence for the first kind of innateness. In one of the most interesting chapters, he argues that the shapes we use to make written letters mirror the shapes that primates use to recognize objects. After all, I could use any arbitrary squiggle to encode the sound at the start of “Tree” instead of a T. But actually the shapes of written symbols are strikingly similar across many languages.

It turns out that T shapes are important to monkeys, too. When a monkey sees a T shape in the world, it is very likely to indicate the edge of an object — something the monkey can grab and maybe even eat. A particular area of its brain pays special attention to those significant shapes. Human brains use the same area to process letters. Dehaene makes a compelling case that these brain areas have been “recycled” for reading. “We did not invent most of our letter shapes,” he writes. “They lay dormant in our brains for millions of years, and were merely rediscovered when our species invented writing and the alphabet.”
ratty  gwern  speculation  language  writing  visuo  eden  dennett  news  org:rec  books  review  summary  roots  deep-materialism 
january 2017 by nhaliday
Goodreads | The Collapse of Complex Societies by Joseph A. Tainter — Reviews, Discussion, Bookclubs, Lists
Very good: much better than Jared Diamond's _Collapse_, and much more convincing than Spengler or Toynbee.
It was also deeply disturbing - the Ik amazed me in chapter 1, and the statistics in chapter 4 were extremely dismal and tie in far too well to Cowen's _The Great Stagnation_ and Murray's _Human Accomplishment_. There are a great many datapoints suggesting that diminishing marginal returns to modern tech/science began sometime in the late 1800s/early 1900s...
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december 2016 by nhaliday
Genome-wide analysis identifies 12 loci influencing human reproductive behavior
- p-values are much lower for women but effect sizes aren't much different?
- actually I think sample sizes are larger so that makes sense
- but there are sex-dependent factors
- FOXP2 makes an appearance, hmm
- Figure 3 is a nice summary of genetic correlations

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december 2016 by nhaliday
The Production of Human Capital in Developed Countries: Evidence from 196 Randomized Field Experiments
Randomized field experiments designed to better understand the production of human capital have increased exponentially over the past several decades. This chapter summarizes what we have learned about various partial derivatives of the human capital production function, what important partial derivatives are left to be estimated, and what – together – our collective efforts have taught us about how to produce human capital in developed countries. The chapter concludes with a back of the envelope simulation of how much of the racial wage gap in America might be accounted for if human capital policy focused on best practices gleaned from randomized field experiments.
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november 2016 by nhaliday
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