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Ask the RD: How Can I Have More Energy and Still Eat Low-Carb? | Nutrition | MyFitnessPal
Vitamin D From a cellular perspective, vitamin D boosts mitochondria — the “batteries” inside our cells that fuel their energy. It also promotes calcium absorption and helps build cells, boost immunity and reduce inflammation. Yet very few foods naturally contain vitamin D; many breakfast cereals, milk and orange juice are fortified with vitamin D — all foods that are either limited or not included in low-carb diets. Good low-carb sources include swordfish, salmon, canned tuna, eggs and sardines. Try including these in your diet more and also going for regular walks outdoors; sunshine helps the body absorb vitamin D.

Magnesium Found in high-carb foods such as beans, whole grains, breakfast cereals, potatoes, rice and legumes, magnesium is required for energy production and regulates protein synthesis and glucose control — all systems that lead to lower energy levels if they’re not functioning properly. Lower-carb foods rich in magnesium include spinach (and other dark leafy greens), almonds, cashews, peanuts, avocado and fatty fish. Adults need between 300–400mg daily, which is why a supplement may be useful if you don’t eat enough of the foods mentioned above.

Choline Best known for its role in brain development, choline also helps build cell membranes, regulate liver function and lower homocysteine levels (high levels in the blood can lead to heart disease and possibly even Alzheimer’s). Pasta, wheat and rice are good sources, but all are high in carbs. Eggs are a great low-carb source, but choline is only found in yolks, so be sure to eat the whole egg. Other good low-carb sources include fish, beef and chicken.

Hydration is key for keeping up energy levels and helping fuel weight loss. Make sure you keep a water bottle on-hand and sip regularly. For an extra energy boost, try adding chia seeds to your water; the powerful seeds contain 10 grams of fiber, 5 grams of protein and 5 grams of omega-3 fatty acids per ounce. They’re also a great source of magnesium.
vitamin  D  magnesium  fiber  micronutrients  choline  flax  seeds  omega-3  fatty  acids  water  hydration  low  carb  carbohydrate 
5 weeks ago by Michael.Massing
Early predictors of metabolic syndrome in healthy 7-9 year-olds identified -- ScienceDaily
Markers that may precede the metabolic syndrome and mechanisms that explain these relationships have yet to be identified. These mechanisms may originate in the intrauterine environment, be exacerbated in susceptible populations, such as African Americans and, be further promoted by a genetic predisposition, particularly among African American children. These abnormalities may work synergistically to create a childhood metabolic syndrome phenotype and may originate earlier in youth than previously proposed.

The purpose of the study was to explore potential correlates of insulin sensitivity, fasting insulin, and insulin resistance, and to determine the best model to predict them in young children. The study of more than 100 healthy children, ages 7-9, found that fat in the liver, abdominal fat, and fat oxidation predicted insulin resistance and appear to be early markers for the metabolic syndrome via a mechanism of impaired lipid metabolism and fat oxidation. Impaired metabolic function may be due, in part, to pre-and post natal factors that are modified by current physical activity. Therefore, race, low or high pregnancy weight and/or birth weight, and low physical activity collectively create a phenotype for poor metabolic function leading to increased risk for insulin resistance in young children.

"Although some of the risk factors cannot be changed, pregnancy weight, birth weight, and physical activity can all be modified and are targets for early intervention to prevent or delay insulin resistance and reduce the risk for metabolic syndrome," noted Dr. Sothern, who is the study's principal investigator.
low  birth  weight  risk  metabolic  syndrome  lipids  fats  blood  cholesterol  skeletal  fat  fatty  liver  epigenetic  insulin  resistance  child  development  children  youth  health  disparities  diabetes  teen  diet  body  disorder  etiology  T2D  research  correlation  type  2  factor  public  genetics  genetic  biomarkers  prognosis  diagnosis  screening  in  vivo  human  clinical  trial  prognostic  leg  prenatal  perinatal  breast  feeding  childhood 
5 weeks ago by Michael.Massing
Insulin resistance/poor insulin sensitivity is closely associated with increased total body fat and may precede development of the metabolic syndrome and type 2 diabetes. Indicators of impaired insulin sensitivity have yet to be clearly identified in children prior to puberty.

The LSUHSC researchers found that the child’s current fat weight is the strongest predictor for poor insulin sensitivity which is a risk factor for type 2 diabetes. LDL (bad cholesterol) was also strongly associated with insulin sensitivity in the prediction model. Previously unidentified Metabolic Syndrome markers discovered by Dr. Sothern’s team include:

• Fat in the liver cells and fat in the skeletal (leg) muscle cells also predict poor insulin sensitivity and high insulin resistance (pre-diabetes) along with an impaired fat burning ability in the muscles.

• These relationships were only found after the researchers considered the child's current fat weight, so the strongest predictor is whether or not these young children are currently overweight or obese.

• The fat in the skeletal muscle became less important after Dr. Sothern’s team considered the mother’s weight prior to and during pregnancy, whether the child was breast-fed, and the current physical activity level of these young children.

“This means that if the mother has a healthy weight gain during pregnancy and the child is breast-fed and physically active, the fat may not accumulate in the skeletal muscle and/or liver and the child may not experience an impaired fat burning ability in the muscle. All of these factors are significantly associated with poor insulin sensitivity that may eventually lead to type 2 diabetes in adolescence or young adulthood. We hope to conduct future prospective studies in this cohort of healthy children to confirm this finding,” notes Dr. Melinda Sothern, LSU Health Sciences Center New Orleans Professor of Public Health and study leader.

Collectively, fat oxidation (how well the body is able to utilize fat as a fuel), blood pressure, and lipids (HDL and LDL) were identified as the best physiologic predictors of insulin sensitivity.

Arlette Soros, MD, an LSUHSC Pediatrics fellow who is a member of Dr. Sothern’s research team, is presenting results of the first study to examine why some children become hypoglycemic (low blood sugar) during insulin sensitivity testing. She will report that children who are lean and have less fat in their skeletal muscle are more likely to get hypoglycemia. Also those with the best insulin sensitivity were the most likely to get low blood sugar.

“We are not sure why this is but think they may be more fit and less prone to diabetes,” concludes Dr. Sothern.
American Diabetes Association 2009 Annual Scientific Session Meeting in New Orleans. Dr. Sothern’s group is the first to document previously unknown markers for obesity, heart disease and diabetes, collectively called the Metabolic Syndrome, in children [ages 7–9]. Posters will be presented on Saturday, June 6, 2009, and Brian Bennett, a Research Associate in Dr. Melinda Sothern’s laboratory will make the oral presentation, Early Markers for the Metabolic Syndrome in Youth, on Monday, June 8, 2009.
low  birth  weight  risk  metabolic  syndrome  lipids  fats  blood  cholesterol  skeletal  fat  fatty  liver  epigenetic  insulin  resistance  child  development  children  youth  health  disparities  diabetes  teen  diet  body  disorder  etiology  T2D  research  correlation  type  2  factor  public  genetics  genetic  biomarkers  prognosis  diagnosis  screening  in  vivo  human  clinical  trial  prognostic  leg  prenatal  perinatal  breast  feeding 
5 weeks ago by Michael.Massing
20 Foods With High-Fructose Corn Syrup (HFCS)
High-fructose corn syrup is seriously bad for your health, but it's being added to all sorts of foods. Here are 20 foods that are often loaded with it.
liver  fatty  fructose 
march 2018 by Jeunj
What Is the Difference Between Sucrose, Glucose & Fructose? | Healthy Eating | SF Gate
Sucrose, glucose and fructose are important carbohydrates, commonly referred to as simple sugars. Sugar is found naturally in whole foods and is often added to processed foods to sweeten them and ...
liver  fatty  fructose 
march 2018 by Jeunj
The Effects of Breakfast Consumption and Composition on Metabolic Wellness with a Focus on Carbohydrate Metabolism. - PubMed - NCBI
Findings from epidemiologic studies indicate that there are associations between breakfast consumption and a lower risk of type 2 diabetes mellitus (T2DM) and metabolic syndrome, prompting interest in the influence of breakfast on carbohydrate metabolism and indicators of T2DM risk. The objective of this review was to summarize the available evidence from randomized controlled trials assessing the impact of breakfast on variables related to carbohydrate metabolism and metabolic wellness. Consuming compared with skipping breakfast appeared to improve glucose and insulin responses throughout the day. Breakfast composition may also be important. Dietary patterns high in rapidly available carbohydrate were associated with elevated T2DM risk. Therefore, partial replacement of rapidly available carbohydrate with other dietary components, such as whole grains and cereal fibers, proteins, and unsaturated fatty acids (UFAs), at breakfast may be a useful strategy for producing favorable metabolic outcomes. Consumption of fermentable and viscous dietary fibers at breakfast lowers glycemia and insulinemia. Fermentable fibers likely act through enhancing insulin sensitivity later in the day, and viscous fibers have an acute effect to slow the rate of carbohydrate absorption. Partially substituting protein for rapidly available carbohydrate enhances satiety and diet-induced thermogenesis, and also favorably affects lipoprotein lipids and blood pressure. Partially substituting UFA for carbohydrate has been associated with improved insulin sensitivity, lipoprotein lipids, and blood pressure. Overall, the available evidence suggests that consuming breakfast foods high in whole grains and cereal fiber, while limiting rapidly available carbohydrate, is a promising strategy for metabolic health promotion.
carbohydate  protein  breakfast  blood  whole  grain  unsaturated  fatty  acids  lipids  UFA  glucose  peer-reviewed  research  response  insulin  fiber  high  low  glycemic  index  correlation  type  2  T2D  effect  diet  self  care  management  long  term  short  viscous  soluble  fermentable  insoluble  metabolism  risk  reduction  harm  prevention  diabetes  metabolic  syndrome 
september 2017 by Michael.Massing
Dietary supplementation of n-3 PUFA reduces weight gain and improves postprandial lipaemia and the associated inflammatory response in the obese JC... - PubMed - NCBI
JCR:LA-cp rats (14 weeks of age) were fed either a control, isocaloric, lipid balanced diet (15% w/w total fat, 1.0% cholesterol, P:S ratio 0.4), a lipid balanced diet with 5% n-3 PUFA [fish oil derived eicosapentaenoic acid (EPA)/docosahexaenoic acid (DHA)] or a lipid balanced diet with 10% n-3 PUFA for 3 weeks. Fasting plasma lipid, cytokine levels, postprandial chylomicron (apoB48) metabolism and the postprandial inflammatory response [haptoglobin and lipopolysaccharide binding protein (LBP)] were assessed following a standardized 'oral fat challenge'.
n-3 PUFA treatment resulted in a significant improvement (i.e. decrease) in the postprandial response for triglyceride (45%) (p < 0.05), apoB48 (45%) (p < 0.03) and LBP (33%) (p < 0.05) compared to controls (measured as area under the clearance curve). In contrast, we observed a significant elevation in postprandial haptoglobin (165%) (p < 0.001) in obese rats supplemented with 10% n-3 PUFA. Treatment with 5% n-3 PUFA in the JCR:LA-cp obese animals resulted in a complementary decrease in total body weight gain (6%) (p < 0.001) and an increase (i.e. improvement) in adiponectin (33%) (p < 0.05) compared to controls, without a concomitant reduction in food intake.
Acute dietary n-3 PUFA dietary supplementation can improve fasting as well as postprandial lipid metabolism and components of the associated inflammatory response in the JCR:LA-cp rat. Further, moderate dose n-3 PUFA supplementation may reduce corresponding body weight during conditions of hypercholesterolaemia and/or modulate inflammation associated with obesity and the metabolic syndrome
fish  oil  omega-3  fatty  acids  EPA  DHA  peer-reviewed  research  overview  risk  benefit  diabetes  triglycerides  dyslipidemia  animal  in  vivo 
march 2017 by Michael.Massing
Effects of eicosapentaenoic acid (EPA) treatment on insulin sensitivity in an animal model of diabetes: improvement of the inflammatory status. - PubMed - NCBI
The major goal of this study was to analyze the effects of fatty acid supplementation on both insulin sensitivity and inflammatory status in an animal model of type 2 diabetes. Diabetic rats (Goto-Kakizaki model) were treated with eicosapentaenoic acid (EPA) or linoleic acid at 0.5 g/kg body weigh (bw) dose. In vivo incorporation of (14)C-triolein into adipose tissue was improved by the ω-3 administration. In vitro incubations of adipose tissue slices from EPA-treated rats showed an increase in (14)C-palmitate incorporation into the lipid fraction. These observations were linked with a decreased rate of fatty acid oxidation. EPA treatment resulted in a decreased fatty acid oxidation in incubated strips from extensor digitorum longus (EDL) muscles. The changes in lipid utilization were associated with a decrease in insulin plasma concentration, suggesting an improvement in insulin sensitivity. These changes in lipid metabolism were associated with an activation of AMP-activated protein kinase (AMPK) in white adipose tissue. In addition, EPA treatment resulted in a decreased content of peroxisome proliferator-activated receptor-α (PPARα) and PPARδ and in increased GLUT4 expression in skeletal muscle. Moreover, EPA increased 2-deoxy-D-[(14)C]glucose (2-DOG) uptake in C2C12 myotubes, suggesting an improvement in glucose metabolism. Concerning the inflammatory status, EPA treatment resulted in a decreased gene expression for both tumor necrosis factor-α (TNF-α) and interleukin-6 (IL-6) both in skeletal muscle and adipose tissue. The data suggest that EPA treatment to diabetic rats clearly improves lipid metabolism although the evidences on insulin sensitization are less clear.
omega-3  fatty  acids  EPA  DHA  peer-reviewed  research  inflammation  benefit  diabetes  triglycerides  dyslipidemia  insulin  sensitivity  in  vivo  animal 
march 2017 by Michael.Massing
Role of ω3 long-chain polyunsaturated fatty acids in reducing cardio-metabolic risk factors. - PubMed - NCBI
Fish oil, rich in eicosapentaenoic (EPA, 20:5ω3) and docosahexaenoic (DHA, 22:6ω3) acids, has been found to cause a modest reduction in blood pressure at a dose level of >3g/d both in untreated and treated hypertensives. Whilst a multitude of mechanisms may contribute to the blood pressure lowering action of ω3 LC-PUFA, improved vascular endothelial cell function appears to play a central role. Recent studies which evaluated the potential benefits of fish oil in type-2 diabetes have helped to alleviate concerns raised in some previous studies which used relatively large dose (5-8 g/d) and reported a worsening of glycemic control. Several meta-analyses have confirmed that the most consistent action of ω3 LC-PUFA in insulin resistance and type-2 diabetes is the reduction in triglycerides. In some studies, fish oil has been found to cause a small rise in LDL-cholesterol, but a change in the LDL particle size, from the smaller more atherogenic form to the larger, less damaging particle size, have also been noted. ω3 LC-PUFA are effective modulators of the inflammation that accompanies several cardio-metabolic abnormalities. Taking into consideration the pleiotropic nature of their actions, it can be concluded that dietary supplementation with ω3 LC-PUFA will lead to improvements in cardio-metabolic health parameters. These fatty acids pose only minor side effects and more importantly, do not interact adversely with the common drug therapies used in the management and treatment of hypertension, dyslipidemia, type-2 diabetes, and obesity/metabolic syndrome, but in some instances work synergistically, thereby providing additional cardiovascular benefits.
fish  oil  omega-3  fatty  acids  EPA  DHA  peer-reviewed  research  overview  risk  benefit  diabetes  triglycerides  dyslipidemia 
march 2017 by Michael.Massing
8. Going by Mai Fatty interview linked below this money was spent by Jammeh Govt in two weeks .…
Mai  Fatty  Gambia  Jammeh  money  from twitter_favs
january 2017 by Digitaldjeli

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